Treating gum disease with homemade remedies

Freedom From Dental Disease

The Primary Care Oral Health Action Pack can Favorably change the way you look at your oral health and what affects it. Increase your knowledge of YOUR teeth and gums, YOUR entire oral environment and yes, your SMILE! Reveal the TRUE CAUSE of cavities, decay of bone and tissue and disclose the proper biological balance. Put YOU in command over this contagious, yet EASILY preventable disease which runs rampant thoughout our population. Allow you to TAKE ACTION to restoring your teeth and gums to optimum health while preventing further decay and damage, as well as preventing any potentially related problems such as heart disease, diabetes, and so on. How to Become Dentally Self Sufficient. Research Advocates OraMedics The science behind the program! The 7 Factors Transcript - Dr. Nara's Last speech before the World Health Federation. Read more here...

Freedom From Dental Disease Summary


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What You Should Know About Gum Disease

These books encapsulate my personal experience with fighting gum disease, described as follows: My gums bled during dental cleanings. I had pockets depths of 4 and 5mms and one 6mm pocket. My dentist told me I had moderate gum (periodontal) disease. She and / or the hygienist also told me that the ONLY thing that would help me was a Scaling and Root Planing Treatment - Otherwise knowns as a SRP or Deep Cleaning. After applying the information I learned on my own and the tools that I employed the results were that my pocket depths returned to normal. There was no more bleeding during dental cleanings, brushing or flossing. I was told that I no longer needed the Deep Cleaning or SRP treatment and that whatever I was doing, I should keep it up. Read more here...

What You Should Know About Gum Disease Summary

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Project Title Periodontal Disease And Recurrent Chd Events

Summary (Adapted from the Investigator's Abstract) This study will analyze the association between periodontal disease and recurrent coronary heart disease (CHD) in individuals who have survived a myocardial infarction (MI). The overall goal is to test the hypothesis that periodontal disease is a significant and independent risk factor for recurrent CHD events. In addition potential mechanisms linking periodontal disease to CHD events will be investigated. At baseline 1,200 participants will undergo a detailed interview and health examination that will include information on lifestyle, sociodemographic, and anthropometric variables and CVD risk factors, and a complete oral health examination including measurement of clinical attachment levels and radiographic assessment of interproximal alveolar crestal height. The two aims are to examine the association between recurrent heart disease and periodontal disease as measured by gingival attachment level and radiographic assessment, and to...

Desquamative Gingivitis

Desquamative gingivitis is a term used to describe a clinical reaction pattern that may be seen in a number of disease processes. It refers to a clinical condition in which the gingiva exhibits atrophy, erosion, and ulceration and possibly a positive Nikolsky sign (27,28). The term is used in a nonspecific manner, as are the terms leukoplakia and erythroplakia, which refer to white and red lesions, respectively, which cannot be attributed to any specific disease process. The designation desquamative refers to the separation of the epithelium from the connective tissue.

Project Title Systemic Effects Of Periodontitis

Summary It is our central hypothesis that periodontal diseases, which are chronic gramnegative infections represent a previously unrecognized risk factor for atherosclerosis and thromboembolic events Previous studies have demonstrated an association between periodontal disease severity and risk of coronary artery disease and stroke. We hypothesize that this association may be due to an intrinsic underlying inflammatory response trait that places an individual at high risk for developing both periodontal disease and atherosclerosis. We further suggest that periodontal disease, once established, provides a biological burden of endotoxin (LPS, lipopolysaccharide) and inflammatory cytokines (especially TxA2, IL- 1B, PGE2 and TNFa) which serve to initiate and exacerbate atherogenesis and thromboembolic events. We propose to test these hypotheses by performing cross-sectional study on 14,000 participants in a longitudinal study of Atherosclerosis Risk Communities (ARIC) to determine the...

Indications For Nutritional Support

The dietary history and using the assumption that maximum requirements of protein and energy for hospitalized patients are 1.5g kg 14h and 40 kcal kg 24 h, respectively. Body composition studies suggest that when the 'finger-thumb test' (feeling the dermis between finger and thumb when pinching triceps and biceps skinfolds) is positive, the body mass is composed of 30 of body protein stores. However a standard clinical history and examination will also identify loss of muscle power, peripheral oedema, skin rashes, angular stomatitis, gingivitis, nail abnormalities, glossitis, paraesthesia and neuropathy.

Gastrointestinal System

Fiber in the diet, and lack of exercise, is more common, leading to increased use of laxatives, Periodontal disease, hemorrhoids, and diverticulosis (inflammation of the walls of the colon) may compound the digestive problems of older adults. Contributing to these functional changes are declines in stomach acid and intestinal secretions, an increase in body fat, changes in the liver and gall bladder, loss of teeth, and a 50 decline in taste buds.

Oral diseases and cariogenicity

Oral refers to the mouth, and includes the teeth and gums (gingival) and their supporting tissues, the hard and soft palate, the mucosal lining of the mouth and throat, the lips, salivary glands, chewing muscles, and upper and lower jaw bones. Digestion begins in the oral cavity, and there are numerous supporting structures for the mouth including the nervous, vascular, and immune systems. Humans contract oral diseases for a number of reasons including genetics, poor hygiene, poor nutrition, alcohol and tobacco use, drug abuse (Shaner et al., 2006), and complications from other diseases such as diabetes (Sandberg et al., 2000, Twetman et al., 2002), cancer (Woo et al., 1993), obesity (Ritchie and Kinane, 2003), and osteoporosis (Norlen et al., 1993). Oral infections themselves may play a role in progressing pathogenesis of many systemic diseases in healthy individuals, ill patients, and those immunocompromised (Ridker et al., 1998). The theory is that oral infections, specifically...

Utility Of The System

As numerous therapeutic drugs and devices are being investigated for their potential benefit in the treatment of periodontal disease, these cell cultures are also being used in the assessment of cell responses and biocompatibility. These developments are of particular interest in relation to tissue engineering. The potential for the cells to differentiate toward an osteoblastic phenotype allows investigation of materials that may be used to There has also been considerable research effort toward the discrimination of cells between the PDL and those from the more superficial gingival connective tissues. This research has been driven by clinical studies suggesting important differences between cells derived from these tissues in leading to the regeneration of the periodontal attachment apparatus. However, differences between these cell types have not yet been clearly defined. Finally, given the unique anatomic role of the periodontium as a protection against local oral bacteria,...

Project Title Endodontic Inflammation And Cardiovascular Outcomes

Summary Several large-scale epidemiologic investigations have uncovered relationships between chronic periodontal disease and adverse cardiovascular outcomes such as coronary heart disease and stroke. To date, endodontic inflammation has not received the same attention despite its being a commonly found sequel to bacterial infection of the Dental pulp space and its having several important characteristics in common with inflammation of periodontal origin. These similarities form the basis of the proposed epidemiologic study, which seeks to test the hypothesis that a greater history of endodontic inflammation is associated with 1) increased risk of coronary heart disease and stroke and 2) increased carotid artery intimal-medial thickness and prevalence of coronary heart disease. These hypotheses will be addressed by linking data from three large, well-established, ongoing epidemiologic studies of aging populations with newly collected variables involving endodontic disease and...

Clinical Manifestations

Hand-Schuller-Christian disease (multifocal LCH) has similar bone granulomas along with other systemic manifestations. The skeletal anatomy of the head and neck is prominently involved. Mandibular defects include severe gingivitis, loss of mandibular height, and multiple loose teeth. The skull can have a geographic skull appearance on plain films secondary to multiple lesions. Involvement of orbital bones can result in changes in vision, and blindness can occur. Sellar involvement around the pituitary can lead to hypopituitarism, resulting in short stature and diabetes insipidus (2).

Upper Airway Colonization

Periodontal disease and dental plaque are clearly identified risk factors for the development of nursing-home acquired aspiration pneumonia. Recent studies have focused on the relationship between oral hygiene, colonization of dental plaques, and subsequent risk of pneumonia (El-Solh et al., 2003 El-Solh et al., 2004). A high plaque index and or evidence of periodontal disease has been associated with presence of anaerobic bacteria in broncho-alveolar lavage (BAL) samples (Imsand et al., 2002). In a study of 49 older patients from chronic long-term facilities requiring intensive care for a lower respiratory tract infection (LRTI), El-Solh et al. (2004) assessed dental status (plaque index, culture of dental plaques) upon admission to the ICU BAL was performed in 14 subjects. The study showed a high rate of colonization of dental plaque by aerobic bacteria (S. aureus 45 P. aeruginosa 13 other GNB 42 ). Furthermore, pathogens recovered from BAL matched the micro-organisms recovered from...

Project Title Periodontal Intervention For Cardiac Events Pilot Trial

Summary (abstract verbatim) This proposal is to develop the clinical trial organization for studies of periodontal treatment in the secondary prevention of cardiovascular events and to develop the institutional teams to accomplish the research. With the appropriate infrastructure and procedures developed, a future definitive randomized clinical trial of the relationship between periodontal infection and cardiovascular disease can be successfully carried out. There is growing evidence of a strong relationship between infection and atherosclerosis as well as a specific link between periodontal infection and heart disease. Previous studies have established possible effective treatments of periodontal disease. These treatments may lead to a diminution of subsequent occurrence of myocardial events in persons at high risk for cardiovascular disease. A clinical trial process to demonstrate the feasibility of a periodontal intervention trial in heart patients requires the establishment of a...

Differential Diagnosis

The differential diagnosis of desquamative gingivitis includes erosive lichen planus (Fig. 28), lichenoid drug reaction, MMP (Fig. 32), and PV (Fig. 37). Often, a positive Nikolsky sign (Figs. 33 and 37) may be demonstrated. The Nikolsky sign indicates mucosal fragility and is seen in diseases with defective epithelial attachment and cell adhesion. In MMP, it is often possible to cause a large flap of gingival epithelium to balloon up (Fig. 33) by blowing a gentle, tangential stream of low-pressure air on an eroded gingival surface. This is surprisingly well tolerated by the patient. Gingival erosive lesions are best treated topically, using a custom flexible tray of the type used for topical fluoride application for dental caries prevention but extended to cover the gingival soft tissues. Linear IgA disease may present as desquamative gingivitis (29). Please refer to the discussion of the individual disease processes that often present as desquamative gingivitis (erosive lichen...

Treatment Complications and Prognosis

NUG generally responds to initial therapy of debridement, antimicrobial oral rinses, and antibiotic therapy (37). When the process heals, the altered gingival anatomy resulting from tissue loss through necrosis creates anatomic forms that predispose to recurrence of NUG and to the development of chronic periodontitis. NUG may progress from gingival soft-tissue involvement to produce loss of periodontal attachment (necrotizing ulcerative periodontitis) and may extend to involve the adjacent oral soft tissues (necrotizing ulcerative stomatitis). Extension of the infectious process through the soft tissues to the facial skin is termed Noma (cancrum oris).

Changes in Inflammatory Cytokines

There has been considerable interest that low grade infections may result in chronic activation of the immune system and that this may predispose to a variety of chronic disease states, including insulin resistance and atherosclerosis. Organisms that have attracted attention include those that cause dental and gum disease, Helicobacter pylori, Chlamydia pneumoniae, and viral infections including cytomegalovirus. A genetic component has also been recognized with polymorphisms in the genes or promoters for many of the cytokines accounting for variation in expression between individuals. For example, the common -174G C polymorphism in the gene for interleukin-6 (IL-6) has recently been linked with longevity as the distribution of genotypes in the very elderly and appears to be different from that of the general population (Hurme et al., 2005). The biological processes involved are obviously very complex, and no single genetic abnormality is going to account for the

FIGURE 1 Linear gingival erythema Source Courtesy of the International AIDS SocietyUSA From Refs 3 4

Also known as HIV gingivitis, a fiery red linear discoloration of the gingival margin can be seen in HIV disease, even without significant plaque formation (Fig. 1). Patients complain of spontaneous bleeding or are asymptomatic. Referral to an HIV dental specialist is recommended and usually involves debridement, local care, and systemic antibiotics. Necrotizing ulcerative periodontitis and stomatitis. Rarely encountered in the asymptomatic HIV-infected individual, this form of periodontal disease occurs in up to 50 of AIDS patients (11). Halitosis is a common complaint, along with severe pain and bleeding. On exam, gingival necrosis of the tips of the interdental papillae with formation of ulcers is found (Fig. 2).

Mucous Membrane Pemphigoid

Erosive lichen planus, MMP, and pemphigus vulgaris (PV) may have similar clinical features. All may present with desquamative gingivitis (Fig. 32). Frequently in MMP, a gentle stream of air blown tangentially at the surface of the involved gingival mucosa will balloon up the epithelium (Fig. 33). They may be differentiated by DIF testing. In MMP, DIF exhibits a smooth linear deposit of immunoreactants (C3 and IgG) along the basement membrane zone. The Nikolsky sign is positive (Fig. 34). Primary herpes simplex is a febrile illness that exhibits an acute onset of vesicles. FIGURE 32 Desquamative gingivitis in mucous membrane pemphigoid. Note the gingival erythema and erosion. Desquamative gingivitis is a clinical reaction pattern that is seen in a number of disease processes, including lichen planus, mucous membrane pemphigoid, and pemphigus vulgaris. FIGURE 33 Desquamative gingivitis in mucous membrane pemphigoid. Note the epithelial separation produced by directing a gentle stream of...

Food and health applications of probiotics translational aspects

Ninety-five percent of the general population has dental caries or periodontal disease (Caglar, 2005). Controlling these diseases has not been highly successful despite preventative therapies such as fluoride and vaccines against oral pathogenic bacteria. Probiotics are now being explored as a treatment option for alleviation or prevention of dental diseases. in cariogenesis. Dental diseases may result due to changes in the complex microbiota present in the oral cavity. Replacement therapy or bacteriotherapy occurs when a pathogenic strain is replaced by a nonpathogenic strain (Caglar, 2005). Bacteriotherapy for S. mutans has resulted in human clinical trials with S. mutans BCS3-L1. This strain was modified to eliminate cariogenicity, promote colonization versus other endogenous S. mutans strains, and limit genetic transformation (Clancy, 2000 Hillman, 1998, 2000). Cariogenicity by S. mutans begins when the bacteria ferments dietary sugars into lactic acid. The decrease in pH due to...

Concluding Remarks

The PDL is an anatomically unique tissue providing functional support for the teeth in the alveolar bone. Although the characterization of the cells derived from this tissue has not provided an absolute marker, the anatomic characteristics do allow for the selection of explant materials specifically from these tissues. The high levels of destructive periodontal disease within the population, leading to the degradation of this tissue, have generated interest in the characterization of cells derived from these tissues.

And Infection

Most infections in patients with severe neutropenia are caused by either aerobic Gram-negative bacilli, particularly Escherichia coli, Klebsiella pneumoniae, and Pseudomonas aeruginosa, or Gram-positive cocci, particularly Staphylococcus aureus and a-hemolytic streptococci (2). In recent years, infections by S. aureus, including methicillin-resistant and vancomycin-resistant species, have been increasing (13). Infections by anaerobic organisms are relatively uncommon, although the microaerophilic organisms of the mouth contribute to gingivitis and mucositis in patients with neutropenia. For unclear reasons, clostridial infections, including bac-teremias, occur in patients with various forms of chronic neutropenia however, clostridial blood stream infections occur infrequently in patients with chemotherapy-induced neutropenia. Clostridia difficile, however, is an important cause of diarrhea, abdominal pain, and fever in all patients with neutropenia, particularly after exposure to...

Viral Infections

CMV disease in HIV is usually reactivation of a latent, previously acquired infection in patients with CD4 count of less than 100 cells mm3 (19) The typical primary CMV infection, presenting with a mononucleosis-like syndrome characterized by fever, pharyngitis, lymphadenopathy, and malaise, is not usually encountered in the setting of HIV infection. In the pre-HAART era, CMV reactivation disease could be asymptomatic or cause fever and constitutional symptoms or end organ disease, occurring in 40 of AIDS patients. The most common organ involved is the retina, in 85 of cases, with the GI tract accounting for another 10 . In the HAART era, incidence of new CMV-reactivation disease has decreased dramatically and is usually seen with CD4 counts less than 50 cells mm3. Oral and esophageal ulcers are the most common upper GI tract manifestation, CMV being the second most common cause of esophagitis after Candida, with HSV being the third. Necrotizing gingivitis and...

Dental Phobia

Around 40 of people with dental phobia have other clinically significant psychological problems such as depression or anxiety (Roy-Byrne etal., 1994). Furthermore, those people have a higher degree of impairment than those with no comorbidity. This is not surprising because more than a third of patients, unselected for dental phobia, are in continual pain when they decide to visit dentists (Green et al, 1997). Those with a phobia of treatment would probably be in more pain because of greater dental disease (Berggren, 1993). Most patients who are too fearful to tolerate dentistry are afraid of experiencing pain that they believe can occur without warning during treatment and they are afraid that they lack control over this (DeJongh et al., 1995 Lindsay & Jackson, 1993). Cognitive interventions would have to address those expectations.



Primary herpetic gingivostomatitis represents the primary infection of the HSV in an individual without prior exposure to the virus, and is accompanied by constitutional symptoms of fever, malaise, and cervical lymphadenopathy. Symptomatic primary infection usually occurs in childhood and is frequently accompanied by painful gingivitis with red, swollen gingiva (Fig. 1). The lesions may affect any mucosal surface, including Both primary herpes simplex and erythema multiforme (EM) exhibit a sudden onset of disease. The lip lesions of primary herpetic gingivostomatitis may bear a resemblance to the crusted lip lesions of EM (Fig. 4). Exfoliative cytology may be useful to differentiate the two by demonstrating the characteristic viral cytopathic effect produced as the epitheliotropic herpes virus replicates within the keratinocytes. Viral culture may also be useful. Lesions of herpangina, caused by the Coxsackie virus, may clinically resemble oral herpes virus infections but typically...

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