Lupus Causes and Treatment

Proven Lupus Treatment

Dr. Gary M. Levin is not a miracle-worker, but a health expert with reputation and expertise that put him at the top of the US medicine field. He has been treating Lupus patients for decades now, and has found that conventional treatments didnt offer real relief, aside from temporarily easing the symptoms. The step-by-step treatment involves Directed Nutrition and a vitamin regime, helps the patient to lose weight, get rid of hair loss and it reduces the constant aches and pain, numbness and tiredness. This Natural Lupus Treatment is addressed first to those who want to learn more about Lupus, and those who are familiar with this area and have a fairly comprehensive knowledge base. Read more here...

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The Lupus-reversing Breakthrough

Heres just a few things youll learn about how to get back into health and conquer Lupus: Those not-so innocent yet everyday substances that are currently attacking your body, perpetuating and aggravating your Lupus. What to do and what Not to do to overcome your Lupus effectively and permanently. How to create the energy you need to be able to work full time and feel confident you will be able to take care of your loved ones. How the pharmaceutical and food industry are conspiring to poison you and make you sick (Hint: American medical system is now the leading cause of death in the US). Which food industries use advertising to encourage doctors to tell you that their food is good for you just like those cigarette ads in the 1950s! The single most effective fruits and vegetables in cleaning up excess acidic waste and how to cleanse your inner terrain completely from systemic acidosis. Why, what your Doctor has told you is wrong, and why many medications actually increase the side effects and complications of Lupus (primarily by depleting vital vitamins, minerals and nutrients from your body). Which supplements every patient must take to decrease inflammation and boost your body's ability to fight Lupus. How to naturally reduce your cravings for toxic foods. Lifestyle and food choices to reverse your Lupus fast, naturally, and for good. Why treating the symptoms of disease is like using an umbrella inside your house instead of fixing the roof. The most powerful creator of health (Hint: its not a food or vitamin!) The best way to simplify the task of making a health-conscious lifestyle adjustment. A miraculous scientific discovery that jump-starts your body to do its natural work, which is to heal itself and restore your Health.

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Lupus Freedom Cookbook

The Only Cookbook In Existence Dedicated Exclusively To Lupus Patients. Hundreds Of Delicious Recipes That Heal. To Be Used In Conjunction With The Lupus Bible & Norton Protocol. This is how The Lupus Freedom Cookbook will change your life: You will never eat a meal that triggers your lupus again. And you very likely did it today. You will gently soothe your endocrine system and shift the ravaging chemical imbalance that is eating away your organs. Kick start the boost of self-healing chemicals that will repair your organs before it's too late. Enjoy delicious meals while knowing every second that you are doing good to your body and getting closer to remission. You won't have to think about where to start in your healing, you will have all the work done for you. When you wake up in the morning you'll feel light and positive, knowing that healing chemicals in your body are doing their work every second. You won't have to spend endless hours in front of your computer or buy nutrition books to know what is completely safe for you. Never again buy another book about diet and health, because you have it all right here and written just for your condition, not general and vague. Start your healing today, without any procrastination. Once again, feel that health and energy you so desperately pursue

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Dog Canis lupus f familiaris

The dog was the first domestic animal however, the beginning of its coexistence with humans is still unclear. The wolf (Canis lupus) is the progenitor of all domestic dog breeds and feral populations. Fifteen thousand years ago, the wolf lived in all of Eurasia, in north Africa, and in North and Central America. It evolved into many subspecies, which differ in size and color. The small Indian wolf (C. l. pallipes) and the larger Eurasian wolf (C. l. lupus) are the most likely dog ancestors.

Human Immunodeficiency Virus and Lupus

Other work has inferred cognitive state from ERP observed in the oddball paradigm. Chao and colleagues (Chao et al, 2004) found that HIVpositive individuals had decreased P200 and 300 amplitudes and longer P300 latencies than controls. Moreover, among HIV-positive patients, depressive symptoms were positively correlated with N1 latency. Similar slowing of components was observed by Jabbari et al (1993). Among HIV-positive patients relative to controls, they suggest that diffuse EEG slowing may be associated with increased reaction time, there may be lower amplitude of visual evoked potentials and brain-stem evoked potentials, and there may be prolonged P300 and N2 latency (Jabbari et al, 1993). The increased latency of N100 and N200 waves in the oddball task was confirmed in a sample with HIV-positive homosexual males and control participants (Bungener et al, 1996). An association was also found between decreased P300 amplitude and emotional blunting. Similarly, in a study of the...

Differential Diagnosis

Multiple diseases can present with findings similar to those seen with Adamantiades-Behget's disease and should be considered when a patient presents with recurrent oral or genital ulcers, inflammatory eye disease, or other manifestations of vasculitis. Included in the differential diagnosis are systemic lupus erythematosus (Chapter 1), seronegative spondyloarthropathies, inflammatory bowel disease (Crohn's or ulcerative colitis) (Chapter 20), herpes or other viral infections (Chapter 10), other forms of vasculitis (Chapter 8), and inflammatory skin diseases such as pemphigus vulgaris or pemphigoid lesions (Chapter 37). All patients presenting with oral and genital ulcerations should undergo testing for herpes simplex virus using culture or polymerase chain reaction methods, to ensure that viral infection is not present. aFindings applicable only in the absence of other clinical explanations (systemic lupus erythematosus, inflammatory bowel disease, seronegative spondyloarthropathies,...

Success and failure of treatment

Low CD4 cells at baseline as well as low viral load before the start of therapy are just two of many factors (Florence 2003, Kaufmann 2005, Moore 2005, Wolbers 2007). Age also plays an important role in older patients, immunological response is often only moderate in comparison to virological response. Several studies demonstrated that the probability of not achieving a rise in the CD4-cell count increases with patient age and with progressive decrease in thymus size as detected by computed tomography (Goetz 2001, Marimoutou 2001, Piketty 2001, Teixera 2001, Viard 2001, Wolbers 2007). Patients who are intravenous drug users also have relatively poor increases in CD4 cells (Dragstedt 2004). In the Swiss cohort, the CD4 cells increased more in women than in men (Wolbers 2007). Other causes for a lack of immunological response may be immuno- or myelosup-pressive concomitant therapies. We have seen patients, who have had a suppressed viral load below 50 CD4 cells l for years, who only...

Chronic Diseases and Neurocognition

Negative cognitive outcomes are also associated with type I and type II diabetes mellitus, pulmonary diseases such as chronic obstructive pulmonary disease and asthma, hepatic diseases such as cirrhosis, kidney diseases, autoimmune diseases such as systemic lupus erythematosus, various cancers, sleep disorders such as obstructive sleep apnea, and the human immunodeficiency virus (HIV) and the acquired immunodeficiency syndrome (AIDS) (see Bellia et al, 2007 Biessels et al, 2008 Borson et al, 2008 Kurella et al, 2005 Tarter et al, 2001 Zhang et al, 2007).

Prey Pursuit and Capture

For an American osprey (Pandion haliaetus) to intercept a fish in shallow water, it must perfectly time its descent and penetration of the water to match the location of its prey. Individual osprey have been observed catching many different kinds of fish, and this suggests that osprey learn to anticipate the position of their prey by observing something about individual fish (Bent, 1961). Numerous predators intercept moving prey (Curio, 1976), whether it is wolves (Canis lupus) taking down moose (Alces alces) in the Yukon or golden eagles (Aquila chrysaetos) catching rabbits (Lepus spp.) in the plains. The behavior seems to be a general one. But does it require an event timer

Influence of FcyRIIa Polymorphism in Infectious or Autoimmune Disease

A few early studies have examined whether expression of the FcyRIIa-Arg His131 polymorphism influences susceptibility to infectious or autoimmune disease. In theory, the weaker binding of human IgG2 to the FcyRIIa-Arg131 variant suggests that this gene might be overrepresented among patients with recurrent infections characterized by certain microbes with polysaccharide coats (i.e., involving an IgG2 antibody response) and overrepresented in disease characterized by circulating immune complexes (because phagocytic cells bearing the FcgRIIa-His131 variant would clear these complexes more readily). Certainly, a skewed genotypic distribution favoring the FcyRIIa-Arg131 variant has been noted in patients with Haemophilus influenzae infections (Sanders et al., 1994) and meningococcal septic shock (Bredius et al., 1994b). Furthermore, there is also predominance of FcgRIIa-Arg131 in patients with elevated levels of immune complexes and glo-merulonephritis complicating systemic lupus...

Internuclear Ophthalmoplegia

Internuclear ophthalmoplegia (INO) results from a lesion in the medial longitudinal fasciculus (MLF) and may be demyelination, tumor, vascular disease, Wernicke's disease, systemic lupus, and others. Lesions at the anterior end of the MLF will have defective convergence. Posterior lesions have better medial rectus function on convergence than on gaze.

Lymphatic and hematopoietic system see also chapters 14 and

Persistent HHV-6 may cause functional disturbances of the immune system as indicated by elevated antibody titers against HHV-6 in allergies, drug-induced hypersensitivity reactions and in systemic lupus erythematosus, Sjogren's syndrome and progressive systemic sclerosis (Krueger et al., 1991, 1994b De Clerck et al., 1992 Klueppelberg, 1994 Lasch et al., 1996 Descamps et al., 1997, 2001 Toh-yama et al., 1998 Conilleau et al., 1999). Virus persistence may constitute a risk factor for additional immune dysregulation and for increasing the severity of adverse reactions (Suzuki et al., 1998).

From the anticonvulsant hypersensitivity syndrome to the DRESS

The diagnosis of this syndrome may be confused with infection, lymphoma, hypereosinophilic syndrome, and collagen vascular disease. Histological examination of skin lesions showed lymphocytic infiltration in the dermis and sometimes the epidermis (epitheliotropism). The infiltrate may resemble the infiltrate observed in the mycosis fungoides (cutaneous T-cell lymphoma) dense band-like infiltrate in the epidermis with atypical lymphocytes. Regarding this lymphocytic infiltration, the biological hematological abnormalities and the clinical lymphadenopathy, pseudolymphoma denomination was also alternatively used for the same patients. The case record of the Massachusetts general hospital reported in 1996 in the New England Journal of Medicine illustrated this discussion (Gorlin and Ferry, 1996). A 7-year-old child was admitted for the association of fever, lymph-adenopathy, hepatosplenomegaly, and eosinophilia. This boy had been receiving carbamazepine and phenytoin for 2 months after a...

Post Surgical Thrombocytopenic Thrombocytopenia Purpura

1-like domains), which is responsible for cleaving ultralarge vWF multimers released from endothelium. Thus, the pathogenesis of idiopathic (primary) TTP likely reflects the formation of arteriolar-occluding complexes of ultralarge vWF multimers and platelets, thereby explaining both the thrombocytopenia and the tissue ischemia. Secondary TTP has been reported to occur in association with pregnancy, certain drugs (e.g., ticlopidine, clopidogrel, quinine, cyclosporine, mitomycin), autoimmune disorders (systemic lupus erythematosus), organ transplantation, and infections (human immunodeficiency virus, bacterial endocarditis). TTP clinically resembles a nephrotropic microangiopathic hemolytic anemia known as hemolytic uremic syndrome (HUS) however, there are certain unique triggers of HUS (especially, preceding infection with E. coli H0157) and anti-ADAMTS13 autoantibodies are not detected in HUS.

Proinflammatory Function For Grp94

In an alternative and intriguing test of the GRP94 cross-presentation hypothesis, the Li laboratory created a transgenic mouse expressing a cell surface form of GRP94 (Liu et al., 2003). These mice were shown to display lupus-like, systemic inflammatory symptoms that correlated with the constitutive activation of dendritic cell function. Interestingly, if dendritic cells from the cell surface GRP94-transgenic mice were adoptively transferred to a mouse of the parent strain, they then display symptoms of chronic systemic inflammation. However, when such dendritic cells were adoptively transferred to MyD88 knock-out mice, no response was seen. Conversely, when MyD88- - dendritic cells were adoptively transferred to the cell surface GRP94-transgenic mice, autoimmune symptoms were observed to resolve (Liu et al., 2003). These data favor the argument that GRP94 is acting principally as an immunological adjuvant, rather than as a cross-presentation antigen and implicate potential signaling...

Interstitial pneumonia in not overtly immunocompromised persons

Totani and co-workers (2001) described an HHV-6-positive interstitial pneumonitis in a 47-year-old woman with Sjoegren's syndrome and Lupus erythematosus. Both autoimmune diseases were previously shown to be accompanied by higher incidences of HHV-6 reactivation (Krueger et al., 1991 De Clerck et al., 1992).

Lepirudin in Pregnancy

A pregnant woman with systemic lupus erythematosus who was treated with dalteparin developed HIT at week 25. Her platelet count dropped from 230 to 59 X 10 L, after which she was treated with lepirudin (15 mg sc twice daily), with aPTT and ECT used to monitor her dosage. Following delivery by cesarean section, she experienced no postpartum bleeding complications, and treatment with lepirudin was continued for several weeks thereafter (Huhle et al., 2000b). Another pregnant woman with lupus anticoagulant and HIT was successfully treated for 36 wk with lepirudin.

Testing Dietary Interventions in Autoimmune Prone Mice to Delay Aging and Age Associated Diseases

The most well-studied autoimmune-prone model examining the impact of CR on immune function is the autoimmune-prone (NZBxNZW)F1 (B W) mouse. This model is especially valuable since multiple organs have been examined, such as spleen, kidney, mesenteric lymph nodes, peripheral blood, and submandibular glands. The B W mouse is a good model to study the human disease Systemic Lupus Erythematosis. As in humans, autoantibodies can be found in young adult B W mice prior to the detection of clinical disease. The B W mice die from autoimmune renal disease (i.e., nephritis), which can be monitored by measuring proteinurea, at approximately 10 to 12 months of age. Feeding the B W mouse a 40 CR diet beginning at six weeks of age delayed autoimmune kidney disease by 30 (Jolly, 2004). The life span of the B W mice could be doubled when the corn oil (CO) based CR diet was substituted with fish oil (FO) (Jolly et al., 2001). It is equally important to note that CR typically does not impact T cell...

Mixed Connective Tissue Disease

Mixed connective tissue disease is characterized by a combination of overlapping features of systemic lupus erythematous, scleroderma, and polymyositis. Typical presentation is with Raynaud's phenomenon, arthralgias, inflammatory myopathy, lymphadenitis, skin or mucosal lesions, and serositis. A key distinguishing factor of the disorder is a high titer of antibody to ribonucleoprotein, a finding absent in any of these three disorders (SLE, scleroderma, and polymyositis). Neurologic dysfunction is present in approximately 10 to 15 of cases, usually presenting with facial pain, facial paresthesias, or aseptic meningitis (96,97). Facial nerve involvement, although far less common than trigeminal nerve involvement, has been described and is felt to be an early manifestation of the disorder (98). Although CSF analysis has suggested an inflammatory involvement of

Affected Organs And Cell Types In Polyomavirusassociated Disease And Persistent Virus Infection

In patients with autoimmune diseases in Taiwan, about 40 were found by PCR to be excreters of polyomaviruses. Interestingly, in 15 , double infections were detected, but none was positive for BKV viruria alone (Chang et al., 1996a). Further extension of the study confirmed the lack of BKV viruria in Taiwan (Tsai et al., 1997). In contrast, patients from Scandinavia with systemic lupus erythematosus had higher levels of BK viruria than healthy control subjects, whereas JCV shedding was in the range of the normal sex-matched control group. In a follow-up study, the authors found a high prevalence of intermittent or even continuous shedding of BKV at 1 year. Immunosuppressive drugs such as corticosteroids, azathioprine, cyclophosphamide, and or metho- Pregnancy is the most common condition of altered immunocompetence that has been linked to Polyomavirus activation (Coleman et al., 1980 Lecatsas et al., 1981). The onset of virus excretion is related to time of gestation, most often...

Heat Shock Proteins As Targets For Immune Cells

Once HSP appear on the cell surface, they are accessible to the immune surveillance system. HSP expressed on cell surfaces can elicit strong immune responses, as these molecules contain several highly conserved epitope sequences with strong immunogenic properties (Kaufmann and Schoel, 1994 Shinnick, 1991 Zugel and Kaufmann, 1999). On the other hand, an overzealous immune response to HSP can have several undesirable effects. Due to high degree of phylogenic conservation, HSP species of microbial origin and HSP molecules produced by stressed host cells have similar immunogenic properties (Zugel and Kaufmann, 1999). Moreover, abundantly expressed HSP undergo processing by antigen presenting cells, and cells expressing HSP alone or presenting them in the context of MHC molecules are recognized by immune cells as potential targets of self-reactive antibodies or lymphocytes with specificity for HSP (Kaufmann and Schoel, 1994 Zugel and Kaufmann, 1999). In a number of autoimmune disorders in...

Case Study Insulin Resistance

The diagnosis of lupus anticoagulant was made based on the physical findings, patient's history, and the laboratory results. Physical finding reveals that the patient had had multiple fetal losses and had pain and swelling in her thigh at the time of medical evaluation. The lack of a positive family history with thrombosis ruled out any inherited throm-botic disorder. Platelet count was normal, indicating that the thrombotic episodes are not related to any cause of platelet activation. aPTT and dRVVT were both prolonged however, the patient did not have any bleeding problems. A prolonged aPTT and dRVVT in the absence of bleeding ruled out any clotting factor deficiency. Mixing study with normal plasma differentiates factor deficiency from an inhibitor. Lack of bleeding rules out factor VIII inhibitor. Lupus anticoagulant is against in vitro phospholipid-dependent tests. In dRVVT confirmatory tests, excess phospholipids are added to the test system to neutralize the lupus antibodies...

HIVassociated nephropathy HIVAN

Experience with other FSGN-forms has shown that only early intervention with HAART - before scarring of the glomeruli occurs due to the underlying disease -has a chance of success. This calls for a rapid reaction HIVAN is independent from CD4 cell count and viral load must be treated The use of components of antiretroviral therapy should take into consideration the different means of renal elimination (adaptation of the dosing). ACE-inhibitors (captropil 6.25 to 25 mg bid, then change to a longer-term effective preparation such as enalapril 5 mg) should be added (see also Table 2). The use of steroids is the subject of controversial discussion (1 mg kg KG day for 2 to 11 weeks), but is favoured in the USA alongside initiation of a HAART, particularly in cases which take a course similar to lupus (Haas 2005, Gupta 2005).

Arrival Of Pml Cases And An Electron Microscope

In the fall of 1962, a particularly stimulating consultation case was presented to me by the pathologist of a downtown Madison hospital. The patient, a 33-year-old woman with lupus erythematosus, had died after several weeks of progressive cerebellar disease. The slides showed a multifocal demyelinating disease with a most striking combination of giant tumor-like astrocytes and large numbers of oligodendrocytes with greatly enlarged nuclei deeply stained with hematoxylin. There were no distinct inclusion bodies as one sees with herpes viruses. I was fascinated and knew I had never seen this disease before. I showed the slides to a visiting neuropathologist and he, too, was at a loss. At that time I was in the midst of a very time-consuming experiment with a group of sophomore students. It involved the induction of brain tumors in chicken with Rous sarcoma virus. There was no time for a library search. However, I did show the slides to Dr. Chou, and to my utter surprise, and delight,...


Conditions such as systemic lupus erythematosus, systemic sclerosis, and insulin autoimmune syndrome (Ito et al., 1993 Crow et al., 1994 Kuwana et al., 1995a). In both lupus and scleroderma, T-helper cells mediate antigen-specific autoanti-body production by B cells (Adams et al., 1991 Mohan et al., 1993 Kuwana et al., 1995b).


Known mechanisms can involve antibody responses to cell surface or matrix components, for example in Goodpasture's syndrome, where the autoantigen is part of the basement membrane. This disease is organ-specific. In other cases, such as systemic lupus erythematosus (SLE), the disease is systemic and multiple tissues and organs may be affected. Immune complexes are thought to play a major role in the pathology of systemic autoimmune disease. A third group of diseases is characterized by T cell destruction of tissues and associated activated cells. This type includes T1D and RA. The classification of autoimmune diseases as organ specific or systemic reflects the underlying etiology. In some families with organ-specific disease several family members may be affected but with different organs involved. Similarly, systemic autoimmunity can affect multiple individuals in a family or a single individual can have more than one systemic condition, such as SLE together with Sjogren's syndrome.


Heparin-induced thrombocytopenia (HIT) is caused by heparin-dependent antibodies that usually recognize multimolecular complexes of platelet factor 4-heparin (PF4-H). HIT can be viewed as a clinicopathologic syndrome (Warkentin et al., 1998). Thus, a diagnosis of HIT should be based on two criteria (1) clinically evident abnormalities, most commonly thrombocytopenia with or without thrombosis (see Chapter 2), and (2) detection of HIT antibodies. In some ways, HIT resembles another clinicopathologic disorder, the antiphospholipid (lupus anticoagulant) syndrome (Table 1).


The human body is a nutrient rich environment and would be colonised rapidly by viruses, bacteria and other parasites if it were not protected by an immune system. This system not only recognises and destroys invading parasites (and even cancer cells), but does not attack the bodies own cells. Both properties are equally important for our survival. An attack of our immune system against our own cells is called autoimmune disease. Such diseases tend to be debilitating, protracted and eventually fatal. Examples are myasthenia gravis (antibody production against the muscle acetylcholine receptor), systemic lupus erythematosus (SLE) (reaction against nuclear antigens) or diabetes mel-litus type 1 (destruction of the -cells in the islets of Langerhans in the pancreas).

Gray wolf

Canis lupus Canis lupus Linnaeus, 1758, Sweden. Twenty-six races are recognized. The largest races live exclusively on large ungulates while the smallest are from the desert regions. Two genetically distinct stocks appear to occur in North America, with wolves in the western part of the continent perhaps representing a separate colonization from Eurasia. Canis lupus H Canis simensis H Chrysocyon brachyurus


This is a self-limiting necrotizing lymphadenitis, possibly of autoimmune basis. Patient presents typically with nontender, persistent cervical lymphadenopathy. Excision biopsy of the enlarged lymph node is required for diagnosis. Some patients may have associated lupus erythematosis therefore, serology for anti-nuclear antibody and rheumatoid factor should be obtained.

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