Natural Sleep Apnea Treatment Systems

The Sleep Apnea Exercise Program

Here Is a Tiny Sample of What Youll Get When You Register for the Sleep Apnea Exercises Program: 18 step-by-step videos that show you exactly how to do the sleep apnea exercises. A 52-page manual that includes a description of each exercise; illustrations to show you how to do each exercise; an explanation of what each exercise does for your body. The manual includes these sections: Causes of sleep apnea; Relationship between sleep apnea and snoring. Scientific studies backing up sleep apnea exercises. How to test your sleep apnea at home. Daily tasks to keep your sleep apnea at a low level. Names and website addresses of speech language pathologists in the U.S. and U.K. who specialize in sleep apnea, and have agreed to list their contact details in my manual. Names and contact details for obstructive sleep apnea support groups. MP3 (audio) recordings of the exercises that you can download and listen to on your iPod, iPhone, or MP3 device. (This is especially useful for the exercises that youll want to do in front of the mirror) Access to an online Members Area, where youll be able to download the manual, watch the videos, and get the bonuses! Read more here...

The Sleep Apnea Exercise Program Summary


4.6 stars out of 11 votes

Contents: 52 Pages EBook, 18 Videos
Author: Marc MacDonald
Official Website:
Price: $47.00

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My The Sleep Apnea Exercise Program Review

Highly Recommended

All of the information that the author discovered has been compiled into a downloadable book so that purchasers of The Sleep Apnea Exercise Program can begin putting the methods it teaches to use as soon as possible.

This ebook does what it says, and you can read all the claims at his official website. I highly recommend getting this book.

Cure Sleep Apnea Without Cpap

In these real-life case studies youll learn in-depth about the lives and treatments of 9 people who have conquered their apnea. Specifically, youll learn: 1. When they first suspected they had sleep apnea. 2. Symptoms that made them first think they had sleep apnea. 3. Steps they took to get diagnosed. 4. How they felt when they were diagnosed (what was going through their mind) 5. The quality of their sleep before their apnea treatment, and how they felt during the day. 6. What they did to try to get a good nights sleep before their successful treatment. 7. What they did to try to overcome fatigue during the day. 8. A description of exactly what their treatment involved. 9. How they found out about the treatment. 10. Side effects of their treatment. 11. Obstacles they encountered during their treatment, and how they overcame those obstacles. 12. How long it took before the quality of their sleep improved. 13. How long it took before they felt better (more rested) during the day. 14. How long its been since they conquered their sleep apnea. 15. Resources they recommend for others who suffer from sleep apnea, and would like to follow their treatment (the name of specific doctors and medical centers) 16. Final words of advice for people who have just been diagnosed with sleep apnea. Here Is a Tiny Sample of What Youll Get When You Download Your Copy Of Cure Your Sleep Apnea Without Cpap: 78 pages of actionable information on alternative, non-Cpap sleep apnea treatments. 9 case studies of men and women who have completely cured their sleep apnea without Cpap. 7 types of alternative treatments that are proven to cure sleep apnea (detailed descriptions) 12 action steps for each alternative treatment, so you know exactly how to take action on each treatment. 7 quick fix sleep treatments that can help you get a better nights sleep Tonight. 69 hand-picked web links for further information on alternative sleep apnea treatments. 31 diagrams explaining alternative sleep apnea treatments

Cure Sleep Apnea Without Cpap Summary

Contents: 78 Pages EBook
Author: Marc MacDonald
Official Website:
Price: $47.00

Easy Sleep Apnea Treatment Singing Exercises

The Singing for Sleep Apnea program provides step-by-step instructions on which singing exercises to follow. Each exercise uses sounds and tunes selected for the strong movements they cause in key areas of your upper airway. The aim of these exercises is pharyngeal fitness: a toned, athletic pharynx that is no longer predisposed to collapse and vibrate in sleep; and which is also wider, resulting in a gentler, less turbulent breath. The exercises are also designed to be fun and to raise your spirits! This complete package includes the following to help you learn the singing exercises as quickly as possible: a) A no-fluff 25-page guide with all you need to know to start your singing exercises, including step-by-step instructions on how to do 13 targeted singing exercises, tips on how to prepare for the singing exercises and how to get the most out of this program (in plain English!) b) 13 instructional audios by noted singing teacher Emily Tucker. These audios have been created specifically for sleep apnea sufferers, and provide step-by-step instruction on each individual singing exercise. c) Bonus ebooks on overcoming insomnia and proven sleep hygiene techniques.

Easy Sleep Apnea Treatment Singing Exercises Summary

Contents: 25 Page EBook, 13 Audios, Bonus EBooks
Author: Marc MacDonald, Emily Tucker
Official Website:
Price: $47.00

Didgeridoo for Sleep Apnea

This complete package includes the following to help you learn to play the didgeridoo as quickly as possible: a) A no-fluff 21-page guide with all you need to know to start playing the didgeridoo including tips on buying the didgeridoo that is right for you, along with advice on how to play the instrument to get maximum benefit (in plain English!) b) 6 instructional videos by noted didgeridoo teacher Tyler Sussman. These videos have been created specifically for sleep apnea sufferers, and provide step-by-step instruction on these topics: History and health benefits of the didgeridoo. How to buy or make your own didgeridoo. Preparing to play. How to make the basic drone sound on the didgeridoo. How to make advanced sounds on the didgeridoo. How to do circular breathing on the didgeridoo. c) Bonus ebooks on overcoming insomnia and proven sleep hygiene techniques

Didgeridoo for Sleep Apnea Summary

Contents: 21 Page EBook, 6 Videos, Bonus EBooks
Author: Marc MacDonald
Official Website:
Price: $47.00

Measuring Sleep Disorders

Sleep architecture can be studied using PSG, and MSLT provides measures of alertness. These studies are not routinely required for the assessment of sleep in PD. However, in cases where obstructive sleep apnea or severe PLM is suspected, PSG is essential. In cases of severe RBD or other parasomnias, PSG is useful for confirmation of diagnosis. A pathological MSLT result (sleep latency < 10 minutes) in a PD patient may also suggest a propensity to sudden onset of sleep.

Rapid Eye Movement Behavior Disorder and Restless Legs Syndrome

The treatment of choice for RBD is clonazepam, a benzodiazepine, although the mechanism is unknown and there are no controlled trials (13). Other drugs thought to be helpful for RBD include pramipexole, levodopa, carbamazepine, donepezil, and melatonin (64,89-91). Caution needs to be exercised with the use of clonazepam, as in some cases, RBD may be confused with sleep apnea, which can be worsened by clonazepam. Nighttime dosing with drugs such as selegiline may aggravate RBD. Others have reported a paradoxical worsening of RBD with deep brain stimulation (DBS) of the subthalamic nucleus (STN) (92).

Chronic Diseases and Neurocognition

Negative cognitive outcomes are also associated with type I and type II diabetes mellitus, pulmonary diseases such as chronic obstructive pulmonary disease and asthma, hepatic diseases such as cirrhosis, kidney diseases, autoimmune diseases such as systemic lupus erythematosus, various cancers, sleep disorders such as obstructive sleep apnea, and the human immunodeficiency virus (HIV) and the acquired immunodeficiency syndrome (AIDS) (see Bellia et al, 2007 Biessels et al, 2008 Borson et al, 2008 Kurella et al, 2005 Tarter et al, 2001 Zhang et al, 2007). Medical and surgical treatments for disease affect cognitive function though in inconsistent directions. For example, prospective investigations generally indicate better cognitive outcomes for those taking antihypertensive medication than untreated hypertensives (Murray et al, 2002). Yet, results of double-blind, placebo controlled trials of antihypertensive have yielded complex and conflicting findings. Statin use may also be...

Setting up mechanical ventilation

Continuous positive airway pressure Continuous positive airway pressure (CPAP) is a continuous positive pressure administered through the ventilator circuit. It is essentially, pressure support (inspiratory positive pressure) plus PEEP (expiratory positive pressure). CPAP provides supplemental pressure to patients who are breathing spontaneously. Patients can be weaned from the ventilator using steadily decreasing levels of CPAP

Project Title Intermittent Hypoxia Cv Impact And Biologic Markers

Summary (provided by applicant) It is estimated that 12-20 million people in the United States have obstructive sleep apnea (OSA), a disorder typified by nightly intermittent hypoxia (IH) exposure due to upper airway obstruction during sleep. Epidemiologic, retrospective, and cross-sectional studies have identified associations between OSA and an increased risk for cardiovascular disease, e.g., stroke, ischemic heart disease, and systemic hypertension. There is emerging evidence that IH may be a more potent sympathetic nervous system stimulus than continuous hypoxia (CH), and that the pattern of hypoxia (repetitive or continuous) is a critical factor in determining physiologic response. Prior studies indicate that the episodic reoxygenation that occurs during IH represents an oxidative stress (OS) that causes cellular generation of reactive oxygen species. Additional studies suggest that OS is likely to be influential in the transcriptional activation of specific genes through which...

Behavioral Medicine and Sleep Future Directions

The development of valid and reliable measures will play a critical role in our ability to evaluate relationships among sleep and health, including the translation of this knowledge to clinical applications. Epidemiologic studies, which play a key role in developing and refining conceptual models, are in need of validated and reliable, low-burden measures that assess traditional and emerging dimensions of sleep, including habitual sleep duration, daytime naps, fatigue, and sleep debt, as well as primary sleep disorders such as insomnia and sleep apnea. These measures may also prove important for reducing participant burden in studies that conduct multiple repeated assessments such as randomized clinical trials and experimental studies designed to probe mechanisms. Wrist actigra-phy holds great promise for decreasing subjective bias associated with self-report measures and for providing high-dimensional data that may be used to probe temporal characteristics of dynamic relationships...

Relationship Between Gh Secretion And Sleep In Pathological States

A few studies have examined nocturnal GH secretion in patients with obstructive apnea before and after treatment (82-84). As expected, nocturnal GH release is decreased in untreated apneic subjects. Because adult patients with this pathology are frequently obese, the low overnight GH levels could reflect the hyposomatotropism of obesity, rather than result from the shallow and fragmented nature of their sleep. However, two studies that have examined the nocturnal GH profile before and after treatment with continuous positive airway pressure (CPAP) have demonstrated that treatment of the sleep disorder resulted in a clear increase in the amount of GH secreted during the first few hours of sleep (83,84). An example is illustrated in Fig. 7. In children, surgical correction of obstructive sleep apnea may restore GH secretion and normal growth rate (82). In obese subjects who do not have sleep apnea, a normal relationship between the first SW episode and GH release may be observed both...

Sleep Abnormalities in Conditions of Deficient or Excessive GH Secretion

In untreated acromegalic patients without sleep apnea, standard polysomnography revealed a reduction in REM sleep as well as a reduction in amount of SW sleep as compared to control subjects (92). However, a more complex picture emerged when power spectral analysis of the EEG was performed (92). Indeed, though the minutes of REM and SW were decreased, the spectral energy per min spent in both REM and SW was increased, indicating that the amplitude of the EEG waves during both SW and REM sleep is higher in acromegalics than in control subjects (92). The amplitude of the EEG is the sum of the post-synaptic potentials of the cerebral cortex, and thus a higher energy presumably reflects an increased neuronal activity in the cerebral cortex. One year after adenomectomy, an important increase in REM time was observed, resulting in normal values for this age group (92). A moderate increase in SW sleep was also observed. Treatment normalized the EEG energy per minute in both SW and REM stages...

Cardiovascular Disease

Several studies examined the relationship between sleep and cardiovascular disease, using polysomnography (Guilleminault et al, 1996 Lee et al, 2008 Taheri et al, 2007 von Kanel et al, 2007 Zaregarizi et al, 2007). The polysomnogram was used to measure factors relating to respiration during sleep, sleep-disordered breathing (snoring, apneas, hypopneas) (Taheri et al, 2007 Lee et al, 2008), and upper airway resistance syndrome (Guilleminault et al, 1996). The polysomno-gram involves the simultaneous measurement of some or all of the following parameters 2-lead EEG, EOG (electrooculogram), ECG (electrocardiogram), EMG (electromyogram), oxygen saturation, and a respiration belt. Apnea is a complete cessation of breathing for at least 10 s, whereas hypopnea is a partial cessation of breathing as derived from the polysomnogram. A number of studies focused on respiratory components of the polysomnogram and factors relevant to cardiovascular disease. Taheri and colleagues (2007) found no...

Long Term Effects of Diabetes

Catterall and colleagues (1984) examined the potential role of diabetic neuropathy, hypothesizing that this may result in sleep apnea, which could explain sudden death in diabetes patients. Polysomnography-derived measures did not differ between the groups and therefore it was concluded that neuropathy-induced sleep apneas are not responsible for sudden unexplained deaths in diabetic neuropathy patients. This conclusion may, however, be deemed premature due to the limited sample size (eight patients and eight controls).


This is usually due to a decrease in airway muscle tone, which may persist up to 5 days postoperatively. In the first 48 h these muscle tone changes are thought to be related to opioids. From about 48 h onwards changes in sleep pattern, not unlike obstructive sleep apnoea, are implicated.


There is normally no pressure gradient at the end of expiration between the airway and atmosphere. Rarely in anaesthesia but often in the intensive care unit, end-expiratory pressure is applied. It is termed positive end-expiratory pressure (PEEP) when ventilation is controlled (Intermittent positive pressure ventilation, IPPV) or continuous positive airway pressure (CPAP) if ventilation is spontaneous.

The Parasomnias

Although clinical history may suggest a diagnosis, in some situations such as when there is a high risk of physical injury or loud snoring suggestive of obstructive sleep apnea, confirmation of diagnosis should be obtained by a single night of polysomnography (PSG) with video telemetry. PSG would show an increased electromyographic (EMG) activity during REM sleep. Symptoms of RBD may predate the diagnosis of PD. Schenck et al. (37) reported that in 1l of 29 men (38 ), 50 years or older in whom idiopathic RBD was diagnosed, a parkinsonian disorder was identified after a mean interval of 3.7 years following the diagnosis of RBD and 12.7 years after the onset of RBD. One study (41) suggested an increased risk of developing PD in individuals who have RBD and olfactory disturbance. This concept is consistent with the recent hypothesis of Braak et al. (21) who suggest that the preclinical stages 1 and 2 of PD start at the olfactory and medullary area of the brainstem. Although the...

And Disease

The overexpression of EPO occurs in a number of adaptive and pathologic conditions. In response to acute hypoxic stress, such as severe blood loss or severe anemia, EPO production can increase 100- to 1000-fold, although the maximal bone marrow response to such stimulation is only a 4- to 6-fold increase in RBC production (46). Overproduction of EPO with accompanying erythrocytosis may be an adaptive response to conditions that produce chronic tissue hypoxia, such as living at high altitude, chronic respiratory diseases, cyanotic heart disease, sleep apnea, smoking, localized renal hypoxia, or hemoglobinopathies with increased oxygen affinity (21). Paraneoplastic production of EPO from tumors and cysts, including renal carcinomas, benign renal tumors, Wilms' tumors, hepatomas, liver carcinomas, cerebellar hemangioblastomas, adrenal gland tumors, and leiomyomas, can also result in high plasma concentrations of the hormone.

Risk Assessment

Risk assessment may involve identification of existing patient physiological risk factors, such as sleep apnea, obesity, and cardiopulmonary compromise such as emphysema or chronic obstructive pulmonary disease. Patients with these risk factors are at much higher risk for developing opioid-induced respiratory depression (128-135). If opioid analgesics will be used, then these patients will require much closer monitoring than comparable patients without these preexisting risks. For all high-risk patients, the best approach is to start low and go slow. However, if aggressive pain management is necessary, such as immediately postop-eratively, then these patients should be placed under constant monitoring for apnea and oxygen saturation, and resuscitation equipment should be readily available.


Treatment should be initiated immediately if there is clinical suspicion. In cases of mild PCP (BGA PO2 > 70-80 mm Hg), ambulatory treatment can be attempted oral medication can even be administered in very mild forms. This may well be possible in cooperation with a competent HIV nursing service. If such monitoring is not possible, if respiratory deterioration occurs, and in every case with resting dyspnea, immediate hospitalization is advised. If ventilation becomes necessary, patients have a poor prognosis, even today. Non-invasive methods (such as CPAP)

Irritant Gases

Treatment includes nebulized racemic epinephrine and systemic corticosteroids. If edema is minimal and early intubation is not required, conservative care to maintain airflow consists of positive pressure breathing (BIPAP or CPAP) or a mixture of helium oxygen gas that due to its lower density can improve upper-airway flow dynamics by reducing turbulence. However, in the presence of edema not immediately requiring intubation, conservative care should only be done if frequent monitoring to assess edema progression and emergent intubation is possible.

Sleep Architecture

Bi-directional relationships among components of sleep architecture and physiological processes important to health and functioning, including metabolic, endocrine, autonomic, and immune mechanisms (e.g., Hall et al, 2004 Opp, 2006 Rasch et al, 2007 Tasali et al, 2008a). In the only study of its kind conducted to date, Tasali and colleagues (2006) demonstrated that selective suppression of slow-wave sleep in healthy, lean adults resulted in marked decreases in insulin sensitivity. Changes in insulin sensitivity were strongly associated with EEG spectral power in the slow-wave delta band and were unaffected by sleep duration. These results are consistent with three observational studies that reported cross-sectional associations among the metabolic syndrome and decreased sleep depth, as measured by decreased visually scored slow-wave sleep or increased EEG spectral power in the fast-frequency beta band after adjusting for age, sleep apnea, and other relevant covariates (Hall, Okun et...


No study summarises the clinical significance of claustrophobia but instances of life-threatening incapacity are described fear and intolerance of radiological examination in an MRI scanner (Mclsaac et al., 1998), of an airway mask for sleep apnoea (Edinger & Rodney, 1993), of gas masks in military personnel (Ritchie, 2002) and hyperbaric delivery of oxygen (Hillard, 1990).

Sleep Duration

Able to recall brief awakenings from sleep and thus may overestimate sleep duration in comparison to actigraphy- or PSG-assessed indices of sleep duration (Hall et al, under review Lauderdale et al, 2008 Owens et al, under review Silva et al, 2007). Subjective bias can be especially evident in populations with significant sleep disturbances, including insomnia, sleep apnea, medical disorders involving pain, and psychiatric disorders (e.g., Rotenberg et al, 2000 Tang and Harvey, 2005). One of the limitations inherent with actigraphy is that the device cannot distinguish between sleep and immobility which reduces its accuracy, although several studies have reported non-significant differences between indices of sleep duration assessed by wrist actigraphy and polysomnography (Chae et al, 2009 Kushida et al, 2001 Hall et al, under review). Finally, it may be important to recognize that measures of sleep duration rarely account for or explicitly evaluate daytime napping. other risk factors...

Sleep Apnea

Sleep Apnea

Have You Been Told Over And Over Again That You Snore A Lot, But You Choose To Ignore It? Have you been experiencing lack of sleep at night and find yourself waking up in the wee hours of the morning to find yourself gasping for air?

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